Created from patient CT and MRI scans. Free, open-source, and accessible.
Pediatric heart models without any anatomical defects. These are a great resource to compare to any of the congenital heart defects
Defects where the left or right ventricle of the heart is under developed, which affects to blood flow throughout the heart.
A type of congenital heart condition that involves the malformation of the aortic arch.
A condition that is a combination of four congenital heart defects. These four defects include: a ventricular septal defect, pulmonary stenosis, a misplaced aorta, and a thickened right ventricular wall.
A condition that, at birth, a child’s 2 main vessels (aorta and pulmonary arteries) that carry blood from the heart are switched.
This is a rare congenital heart defect whereby the pulmonary veins carrying the oxygen-rich blood from the lungs do not connect to the left side of the heart.
Heart models with multiple defects, or a more complex and difficult to categorize diagnosis.
We are working hard preparing more heart models for the library!
In the meantime, click on the link below to explore more heart models on our Sketchfab page
A child with a coarctation of the aortic arch after the brachiocephalic artery. The narrowing (coarctation) of the aorta makes it harder for the heart to pump blood to the body. This can lead to complications including not getting enough blood to the body’s organs. Babies can have weak pulses in their legs or cold legs, trouble feeding and gaining weight, rapid breathing, pale skin, and sweating.
explore modelThis is a model of a patient with an unbalanced atrioventricular septal defect (AVSD) with a hypoplastic left ventricle who underwent a Fontan procedure as the final stage of the surgical palliation. This is a myocardial model, meaning that you are seeing the muscle of the heart rather than the blood inside the heart. This model has been coloured to demonstrate the presumed oxygen saturations within the different chambers and blood vessels of the heart in the setting of a Fontan fenestration. You can see that there is a small amount of deoxygenated (blue) blood that is able to travel from the Fontan conduit into the right atrium through the Fontan fenestration.
explore modelHypoplastic Left Heart Syndrome (HLHS) is a heart condition where the mitral and aortic valves don’t develop properly. The left ventricle and aorta are also underdeveloped and very small (hypoplastic). This prevents oxygenated blood from reaching the body. Instead, blood is forced through a hole (an atrial septal defect) from the left atrium to the right atrium with blood reaching the aorta though the pulmonary artery and ductus arteriosus (a connection between the aorta and pulmonary artery) to reach the body. There can be other differences in the heart that can occur with HLHS.
explore modelHypoplastic Left Heart Syndrome (HLHS) post Glenn, pre-transplant assessment. This coloured bloodpool model shows the mixing of blue deoxygenated blood and red oxygenated blood. Colouring is not based on scientific data. In hypoplastic left heart syndrome, the left ventricle is very small. The Glenn surgery connects the superior vena cava (SVC) to the pulmonary arteries to allow the deoxygenated blood returning from the upper body to bypass the heart and travel to the lungs directly.
explore modelHypoplastic Left Heart Syndrome (HLHS) with mitral stenosis and aortic atresia. This patient had a Norwood/Sano operation. This model is coloured to show the deoxygenated (blue) blood and oxygenated (red) blood, which mixes within the heart (indicated as purple). The colouring is meant to explain the physiology and blood flow in a heart after a Norwood/Sono procedure.
explore modelInterrupted Aortic Arch (IAA) is a rare congenital heart defect where the aorta (the body’s main artery) is completely disconnected, preventing oxygen-rich blood from reaching beyond the gap(the lower body). Before and shortly after birth, a temporary vessel called the ductus arteriosus bridges this gap. When the ductus arteriosus begins to close after birth, it leads to severe, life-threatening symptoms because the lower body and organs are deprived of oxygen-rich blood. Many babies with IAA also have a Ventricular Septal Defect (VSD), a hole between the heart’s lower chambers.
explore modelPulmonary atresia means that there is no connection between the right ventricle and the pulmonary artery, which prevents blood from flowing out of the right ventricle and to the lungs. Instead, blood is forced through a hole (an atrial septal defect) between the top two chambers (atria) to send deoxygenated blood to the left side of the heart. The deoxygenated blood travels through the aorta and ductus arteriosus (a connection between the aorta and pulmonary artery) to reach the pulmonary artery and lungs. The right ventricle is often underdeveloped and very small (hypoplastic). Babies born with this condition need a way for the de-oxygenated blood to reach the lungs and pick up oxygen.
explore modelPulmonary atresia means that there is no connection between the right ventricle and the pulmonary artery, which prevents blood from flowing out of the right ventricle and to the lungs. There is also a hole present in the ventricular septum (ventricular septal defect or VSD). Blood flows from the right ventricle to the left ventricle and the deoxygenated blood can leave the heart through the aorta. The deoxygenated blood travels through the aorta to the lungs through the ductus arteriosus (a connection between the aorta and pulmonary artery).
explore modelThis is a 3D model from a neonate with pulmonary atresia, ventricular septal defect (VSD), and major aortopulmonary collateral arteries (MAPCAs). This patient has 3 MAPCAs which are seen as the abnormal vessels arising from the aorta to supply the lungs. In this patient these vessels have areas of narrowing (stenosis). In this type of congenital heart disease, the patient is born without a pulmonary valve leaving no connection between the heart and the pulmonary arteries. The MAPCAs are formed by the body to supply blood to the lungs. The VSD allows blood to mix between the right and left ventricle.
explore modelThis model demonstrates the anatomy of a right aortic arch with aberrant left subclavian artery leading to a vascular ring, which can cause airway and esophageal compression. In this model, the right sided heart structures are depicted in blue, the left sided heart structures in red, the airway in white, and the esophagus in yellow.
explore modelTetralogy of Fallot (TOF) is a set of four changes to the heart that include narrowing of the pulmonary artery (pulmonary stenosis), a hole in the ventricular septum (ventricular septal defect or VSD), the aorta moving towards the right side of the heart (“overriding aorta”), and thickening of the right ventricle (right ventricular hypertrophy).
explore modelIn total anomalous pulmonary venous return (TAPVR/TAPVD), the pulmonary veins do not return to the left atrium. In the “cardiac” type, the pulmonary veins return to the heart to drain into the right upper chamber (right atrium). The only way for blood to get to the left side of the heart is by crossing through a hole (an atrial septal defect or ASD) between the top two chambers (atria) to reach the left side of the heart.
explore modelThis is a complex heart lesion that combines many heart defects. Diagnosis includes: Complete atrioventricular septal defect (AVSD), major aortopulmonary collateral arteries (MAPCAs), infradiaphragmatic total anomalous pulmonary venous return (TAPVR), hypoplastic left ventricle, right aortic arch, single coronary artery.
explore modelTransposition of the Great Arteries (TGA) occurs when the two main arteries or blood vessels leaving the heart are reversed. The pulmonary artery arises from the left ventricle (instead of the right), sending oxygen-rich blood back to the lungs. The aorta arises from the right ventricle (instead of the left), sending oxygen-poor blood back to the body. This misrouting stops oxygen from being delivered to the body’s tissues and organs and can be life-threatening. The body does not receive the oxygen it needs to function properly. Babies can look blue, especially around the lips and nails. Babies can have a fast heart rate, fast breathing, and poor feeding.
explore modelThis is a model of a normal teenage heart with no defects. It has been coloured to show the two halves of the heart - blue representing the deoxygenated blood and red representing the oxygenated blood. In this model, you can also see the outline of the heart muscle (myocardium) around the ventricles and atria.
explore modelAn unbalanced AVSD (the left ventricle is very small) and Fontan physiology. The Fontan procedure connects the inferior vena cava (IVC) to the right pulmonary artery. Now all the deoxygenated blood from the body bypasses the heart and directly enters the lungs. There is also a small hole between the Fontan conduit and the right atrium which is called a fenestration. The fenestration allows a small amount of the blue deoxygenated blood from the Fontan conduit to enter the heart where the red oxygenated blood is located. This fenestration is meant to relieve some pressure by acting as a "pop-off valve" until the lungs adjust to the new circulation.
explore modelThis is a model of a patient with an unbalanced atrioventricular septal defect (AVSD) with a hypoplastic left ventricle who underwent a Fontan procedure as the final stage of the surgical palliation. This is a myocardial model, meaning that you are seeing the muscle of the heart rather than the blood inside the heart. This model has been coloured to demonstrate the presumed oxygen saturations within the different chambers and blood vessels of the heart. In this model, there is no Fontan fenestration present.
explore modelThe 3D Heart Project is a growing initiative—we are a multidisciplinary team and always welcome new collaborations.
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